If you are asking if that device has any significant permanent positive effect on wild-type amyloidosis, the answer is no.

Amyloid fibrils are proteotoxic. Proteotoxicity refers to the adverse effects of damaged or misfolded proteins and even organelles on the cell. At the cellular level, the ultimate outcome of uncontrolled or severe proteotoxicity is cell death; hence, the pathogenic impact of proteotoxicity is maximally manifested in organs with no or very poor regenerative capability such as the brain and the heart.

If you can't remove those fibrils from your heart or other areas of your body, the disorder is an ongoing destructive force that ultimately kills you as your heart fails due to total electical and mechanical failure. No device can correct the total loss of electrical conductivity and loss of ventricular muscle response (contraction). So, no, I think an appropriate pacemaker , medication, and electrophysiologist intervention is the most helpful.